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Mechanistic study of BNP7787-mediated cisplatin nephroprotection: modulation of human aminopeptidase N
Crescenz VA Medical Center, Philadelphia, PA, (3)VA Informatics and Computing Infrastructure, (4)VA Informatics and Computing Infrastructure (VINCI), (5)University of Pennsylvania, Philadelphia, PA, (6)Division of Gastroenterology and Hepatology, Department of Medicine, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA, (7)University of Pennsylvania, (8)Department of Medicine, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, (9)VA Long Beach Healthcare System Background: Nonalcoholic fatty liver disease (NAFLD) is a leading cause of chronic liver disease
According to studies, NAC may help lower elevated levels of homocysteine in the body, helping convert this amino acid into its active proceeding form
There is substantial evidence confirming that excessive ROS acts as common apoptosis-inducing factors, with mitochondria serving as the primary pro-apoptotic target [35]
