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As previously established, the liver relies heavily on glutathione conjugation (via Glutathione S-Transferases) to neutralize and excrete toxic intermediates, including excess inflammatory mediators and circulating histamine
[1] [3] The compounded nature of this product allows for individualized dosing and administration, which may be advantageous in clinical scenarios where standardized commercial products are not available or appropriate
Figure 3: Changes in the lameness period amongst the four experimental treatment groups
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