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The large number of metabolites such as QA generated under this condition can pass through the damaged BBB and enter the CNS, further intensifying neuroinflammation and oxidative stress, and lowering the threshold for epileptic seizures [14, 15]

1,2 By specifically binding to the key hormone GLP-1, it regulates blood glucose levels and lipid metabolism

In cell and tissue models, GHK-Cu has been associated with upregulation of genes tied to collagen types I and III, elastin, decorin, several matrix metalloproteinases and their inhibitors, and pro-angiogenic factors such as VEGF and FGF2

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