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[1] [2] B12 functions as a cofactor in cellular metabolism, participating in the conversion of homocysteine to methionine and the metabolism of methylmalonic acid, both crucial for energy production
Follicle miniaturization in androgenetic alopecia is associated with reduced perifollicular vascularization, and interventions that increase VEGF expression may help maintain the blood supply that follicles need to sustain robust growth
Medications that interfere with B12 absorption (e.g., metformin, proton pump inhibitors)
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