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Through its connections with the hypothalamus and limbic system, the brainstem also influences neuroendocrine and immune pathways, including the HPA axis and inflammatory signaling

The trial included 80 people randomized to 0.1 mg of semaglutide, 78 to 0.2 mg, 82 to 0.4 mg, and 80 to placebo [3]

Accordingly, excess DPP-4, derived from adipocytes and/or hepatocytes, may act as a local mediator of inflammation and adipose/hepatic tissue insulin resistance, thereby forming one link between obesity and the pathogenesis of T2DM and metabolic disease (Figure 3)

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