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This review synthesizes current research on ferroptosis mechanisms in CHD and emerging clinical intervention strategies
However, loss of DJ-1 promotes the dissociation between p65 and NF-B inhibitor (IB), facilitating p65 nuclear translocation, resulting in more microglial activation and aggregation of microglia-mediated neurotoxicity in an NF-B-dependent manner [137]
Numerous studies have shown that GSH anabolism and GPX4 are inextricably linked to ferroptosis
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