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Figure 2 Glycocalyx disruption, platelet activation, and nitric oxide pathway dysfunction in the pathophysiology of delayed cerebral ischemia In a small case series, DCI was associated with significant elevations of soluble markers indicative of endothelial injury, including vascular cell adhesion molecule 1 (VCAM-1) and other cell adhesion molecules, along with increased levels of syndecan-1, a specific biomarker of glycocalyx degradation

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Nesfatin-1 crosses the blood-brain barrier without saturation

doi:10.1016/j.ejim.2021.01.002 Sumithran P, Prendergast LA, Delbridge E, et al