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Estrogen has been shown to upregulate endogenous MOTS-c expression in preclinical models, which may partly explain why metabolic deterioration accelerates in perimenopause when estrogen falls precipitously [2]

This is not fringe science it is increasingly mainstream in ageing and redox biology research, even if supplement marketing has not caught up

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The successful execution of canonical pyroptosis in SUP-B15 cells underscores that specific cellular contexts are highly susceptible to X(PC)-induced pyroptosis