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Triggered by mitochondrial oxidative stress, Ca 2+ overload, dysregulation of iron homeostasis, mitochondrial DNA defects, and disruption of mitochondrial quality control (MQC), programmed cell death subsequently ensues encompassing apoptosis, pyroptosis, necroptosis, and ferroptosis [17]
pneumoniae lacks the capacity for endogenous GSH synthesis
Agilent Technologies)
Though rare, some individuals react adversely to copper compounds
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