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Hypothalamic amylin receptor activation reduces food intake through complementary but distinct circuits from GLP-1 receptor-mediated satiety signaling

I think many of us nodded along thinking that Terrys joke that there were more authors on the subsequent paper (Pickering et al, Kidney Int 2013) than patients with the disease, would mean we wouldnt have to re-educate ourselves on the labyrinthine links between old fashioned MPGN, new fangled C3G, and everyones least memorable bit of basic biology (well, maybe after the coagulation cascade), the complement system

doi:10.1002/anie.199502591 Return to citation in text: [1] Noyori, R

For most people, the answer provides welcome relief