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[16] The principal AOD9604 studies cited here did not establish a complete AOD9604-to-UCP1 pathway or demonstrate direct induction of UCP1 transcription, UCP1 activity, white-adipose browning, or functional thermogenesis

Mice tolerated doses several times higher than the effective dose with no observable adverse effects

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Because this region has been linked to fat metabolism in research models, it is sometimes described as part of a lipolytic domain. In laboratory settings, AOD 9604 is used to explore how growth hormonederived fragments may influence fat breakdown, fat storage, and energy use in cell-based systems and non-clinical animal modelswithout the broader hormonal activity of full-length growth hormone