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Mapping the signature genes of the NR3C1 CD4 subset to pathway gene sets showed that TGF signaling is among the top upregulated pathways, suggesting that TGF signaling might be critical for either the function or maintenance of this T cell subset (Fig
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The substitution that does the real work is D-Ala2, which blocks cleavage by dipeptidyl peptidase-4, the protease that degrades native GHRH within minutes
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