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GSH depletion can trigger ferroptosis, which has been exploited in cancer treatments [17], and supplementation with GSH is utilized by glutathione peroxidase 4 (GPX4) to prevent lipid peroxidation, in turn inhibiting ferroptosis

As a core transcription factor regulating the cellular anti-ferroptosis program, Nrf2 inhibits lipid peroxidation and the accumulation of labile iron by inducing the expression of its target genes, demonstrating significant potential in counteracting ferroptosis (71)