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Furthermore, oxidative stress cascades involving ROS, neuroinflammatory pathways such as TLR-4, NFB, and NLRP3 inflammasome-driven pyroptosis, vascular endothelial growth factor signaling, as well as diverse forms of cell death, including organellar dysfunction, autophagy, apoptosis, ferroptosis, and necroptosis, require further research to assess their potential as effective therapeutic targets (130)

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Acknowledgements This work was supported by Chang Gung Memorial Hospital [CMRPG391311, CMRPG3A1131, CMRPG3H0611 and Grants CIRPD1D0071-3