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Chronic sleep deprivation disrupts leptin and ghrelin balance, increasing hunger signals and reducing satiety responses
Studies suggest a role for brain-derived GLP-1 in the control of eating and reward (80, 81), but there is no evidence that peripherally administered GLP-1RAs can directly access GLP-1Rs in these regions, as demonstrated by the lack of fluorescently labeled semaglutide in these brain nuclei
Ongoing monitoring and support is considered in the context of your overall health plan, clinical eligibility, and long-term goals
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