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GLP-1 inhibits intestinal contractions through mechanisms that may involve the central nervous system via vagal pathways or direct action on central receptors [24], as well as the enteric nervous system by modulating neurotransmission through presynaptic receptors affecting nitric oxide release [25,26]
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18 TGF- functions through its interaction with TGF- receptors, which phosphorylate SMAD2/3, allowing these proteins to form complexes with SMAD4 that translocate to the nucleus and regulate gene expression
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