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Description

its expression is the highest in alveolar type II cells

Sequence:39-amino-acid synthetic triagonist peptide (fatty-acid-modified) Origin: Engineered triple incretin analogue developed as a single-molecule agonist at GIP, GLP-1 and glucagon receptors

Intestinal farnesoid X receptor signaling promotes nonalcoholic fatty liver disease

The 6.0 mg dose showed slightly less MASH resolution than 4.8 mg, likely because higher dropout rates from side effects affected the intent-to-treat analysis