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Toxics , 3(1), 20-62

We think this could lead to new interventions, such as reducing the glutathione overload through synthesis inhibitors or compounds that can dissipate it. The findings also have implications for potential therapies for lung cancers related to mutations in the KEAP1 gene

Bayesian models forecast that although prevalence may plateau or modestly decline by 2040, mortality and overall disease burden will continue to rise, driven largely by population aging, especially in high-income countries [1] and increased environmental toxic compounds

In translational neuroscience, reliably demonstrating neuroprotection in humans remains particularly difficult, not only due to technical limitations but also because of this enduring conceptual imprecision