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A mechanistic study has further implicated lactate in ferroptosis-mediated lung injury during sepsis progression (82)

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albicans systemic infections by enabling the hyphae to penetrate endothelial tissue and subsequently seed the bloodstream with yeast cells 1,2,3

These issues include the absorbance of the drug overlapping with the absorbance of the mAb as well as the interference caused by the presentence of high levels of non-covalently bound free drugs, leading to over-quantitation of the drugs and relatively high average DAR values