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Prescription trends illustrate this shift

It seems that relatively lower IR protects glucose metabolism in PWS, and adipose tissue, adiponectin, ghrelin, oxytocin, irisin, growth hormone and ANS all may play a role in lower insulin levels and in lower IR in PWS patients (summarized in Fig

When cardiolipin becomes oxidized, it migrates to the outer membrane, where it can interact with cytoplasmic amyloidogenic proteins to facilitate the formation of pores in the mitochondrial membrane, and, ultimately, to trap mitochondrial debris from aged mitochondria within fibrillar extracellular deposits such as Lewy bodies (Seneff et al., 2025b)