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In conclusion, we show that APAP toxicity in HepG2 cells is largely independent of oxidative stress, and is linked instead to a decoupling of glycolysis from the TCA cycle, lactic acidosis, reduced NADPH production, and subsequent suppression of the anabolic pathways required for rapid growth
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Moreover, it looks like a step toward delivering efficacy comparable to bariatric surgery, but with drug therapy
Additional metabolites displayed qualitative differences in abundance across groups but did not reach statistical significance
However, this suppression appeared to be lessened when Ipamorelin was combined with ghrelin
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