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Of note, neuroinflammation has furthermore been implicated in the pathophysiology of depression [87], whereas increased inflammatory markers, such as C-reactive protein (CRP) or interleukin-2 (IL-2), have been specifically described to be more associated with the atypical subtype of depression [69]
Conversely, knocking out the gene ECE1 that only encodes candidalysin results in a sharp decline in IECs damage, barrier disruption and penetration ability 112
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