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Furthermore, exenatide has been demonstrated to mitigate mitochondrial oxidative stress, reduce Ca 2 overload, inhibit fission, and close the mitochondrial permeability transition pore (mPTP), collectively preventing cardiomyocyte apoptosis by inhibiting the release of proapoptotic proteins such as cytochrome c and caspase-3 [94]
4,17 Both GIP and GLP-1 mediate their actions by binding to their specific GPCRs on target tissues: GIP binds to the GIP receptor (GIPR) and GLP-1 binds to GLP-1R
lucidum (Reishi) and C
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