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It suggests that substantial therapeutic value may reside in revisiting known peptide systems with modern tools, rather than focusing exclusively on the discovery of entirely new ligands

While this initially induces superoxide release from mitochondria to cytosol, the subsequent activation of the MAP kinase JNK and its translocation to mitochondria amplifies mitochondrial oxidative and nitrosative stress, facilitated by lysosomal iron transfer

The GLP-1R:TT-OAD2 interactions modify the hydrogen bond network between TM1 and TM2