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The onset of cardiovascular disease (CVD) is proposed to be mediated by a multitude of drivers, like metabolic stressors, cardiac and vascular dysfunction, impaired angiogenesis, and the development of atherosclerosis [2]
The rationale is that even sub-therapeutic doses activate the GLP-1 and GIP receptors enough to produce noticeable appetite modulation without the gastrointestinal side effects that trouble many users during standard initiation
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