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Patients with NAFLD exhibited a significantly lower total bacterial load compared to normal subjects, accompanied by a reduction in the abundance of various normal bacteria in the intestinal wall, including Bifidobacterium, Lactobacillus, Lactococcus, True bacillus and Propionibacterium (Vakhrushev et al., 2022)

Collectively, these findings identified EV-derived VDAC1 transfer as a novel therapeutic target for overcoming PTX resistance in GC, highlighting the dual role in modulating both mitochondrial homeostasis and autophagic flux

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