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63, 64 Interestingly, the presence of proinflammatory cytokines and activated neuroglia have been reported in the autism cerebellum among other regions, 9 suggesting that activated neuroglia produce excess NO and may contribute to the peroxynitrite formation and increased 3-NT protein damage observed in the present study
PRKAR1A mutations and loss of PRKAR1A protein expression are seen in most cases and should prompt a search for other findings of the Carney complex [3, 4]
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