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This range may be optimal, according to some research, given its association with improved cognitive function
Because these were also the polyunsaturated fatty acids that were lost during LPO [78] , these observations suggest that this diet substantially enhanced the susceptibility to LPO and liver injury
Molecular docking revealed enhanced binding activity for HT with targets related to oxidative stress, including Nrf2, NAD(P)H quinone oxidoreductase 1 (NQO1), thioredoxin reductase 1 (TrxR1), prostaglandin-endoperoxide synthase 2 (PTGS2) and aldo-keto reductase 1C3 (AKR1C3)
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