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The mechanism behind it remains unknown
It is reported that 5-ASA ameliorates UC in mice through dual mechanisms of preserving intestinal epithelial barrier integrity and modulating gut microbiota composition, with concomitant suprpression of JNK and p38 MAPK phosphorylation in colonic macrophages (Qu et al., 2017
ROS generation is controlled under physiological conditions but increases under pathological conditions, with high ROS levels potentially inducing carcinogenesis and oxidative stress-stimulated diseases
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