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This oxidative and nitrosative stress further damages mitochondria, lowers the apoptotic threshold, and creates a self-reinforcing cycle of inflammation and cell death [93]
Levels of this natural peptide in the human body decline sharply with age, which is closely associated with the gradual deterioration of the organisms regenerative capacity
The iNOS-deficient M2 phenotype exhibits high sensitivity to iron-containing stimuli, a sensitivity that is completely eliminated by the exogenous addition of NO donors [62]
to reduce the risk of major cardiovascular events such as heart attack, stroke, or death in adults with type 2 diabetes with known heart disease
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