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GHK-Cu is generally well-tolerated, but some individuals may experience mild or temporary side effects: Less common side effects may include: Most side effects are mild and resolve on their own

29, 30, 31, 35 This results in an improved energy metabolism, which counteracts possible cell death processes activated at mitochondrial level, as also suggested in a previous work on an analogous acetylated molecule

It should be noted, however, that ALDH1A1, but not CYP2E1, is a downstream target of NRF2, which was found to be activated in untreated GCLM KO mouse liver

Gene therapy as a curative option for Beta-thalassemia