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Its approval was supported by data from the Phase III CAHtalyst study, which demonstrated that Crenessity significantly lowered androstenedione concentrations in both adult and pediatric patients, allowing them to taper glucocorticoid doses without affecting treatment efficacy
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Although initial studies suggested that GSH depletion was only a byproduct of oxidative stress generated during cell death, recent discoveries suggest that GSH depletion and post-translational modifications of proteins through glutathionylation are critical regulators of apoptosis
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