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The nuclear factor-kappa B pathway has long been considered a prototypical proinflammatory signaling pathway, largely based on the role of NF-B in the expression of proinflammatory genes including cytokines, chemokines, and adhesion molecules 20

(Wounds, 2003) documented GHK-Cu's effects in animal wound models, while additional studies have examined its role in bone remodeling, nerve tissue research, and hair follicle biology