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In the current study, depriving RPE cells of the essential GSH precursor Cys 2 by culturing cells in Cys 2 -free culture medium or blocking de novo synthesis of GSH by using GCL inhibitor BSO or blocking Cys 2 uptake by pharmaceutical inhibition of system x c caused cell death
This activation promotes blood vessel formation during healing
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Enhanced expression of defense-related genes like GSTs during systemic infections could be also due to the silencing suppressor activity of the infecting virus, as shown for A
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