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Mikhailova TL et al
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Unlike full-length growth hormone, which is associated with alterations in insulin sensitivity in experimental systems, AOD 9604 demonstrates a distinct signaling profile [2]
IGF-1 and growth hormone receptor activation controls confirming that AOD-9604 does not engage the classical hGH receptor or elevate IGF-1, distinguishing its mechanism from full-length growth hormone in cell-based reporter systems
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