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Furthermore, modulation of endogenous incretin hormones (GLP-1 and GIP) could be an interesting strategy for preventing and/or managing type 2 diabetes mellitus (T2DM) (Reference Kreymann, Williams and Ghatei8)

These recruit the most muscle and produce the strongest metabolic stimulus

Given pulmonary surfactants pivotal role in RDS pathophysiology and the dependency of palmitoyl-carnitine synthesis on adequate L-carnitine availability, strategies to augment L-carnitine levels represent a biologically plausible approach to support surfactant production and potentially ameliorate RDS severity

These reactions can be worse when glutathione is given through IV