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The decrease in the expression of the ATP-F1- subunit, probably due to epigenetic changes related to hypermethylation of the gene, has been reported in cells from refractory or relapsed AML patients and has been confirmed in DOX-resistant HL60 myeloid lines and DNR ( Mitochondrial fusion and fission are two opposite processes that go according to cellular metabolic requirements
Arch Biochem Biophys (2021) 706:108926
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