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We showed that cirrhotic (an IGF-1 deficiency condition) rats had diminished amino acid and glucose intestinal absorption [114, 194, 195] and that IGF-1 replacement therapy was able to restore both alterations, suggesting a role of IGF-1 in the position of transporters [196]
Since rodent and human -cells express DA D 2 -like receptors 22,23 , APDs may also directly target -cell D2R/D3R to raise glucagon and drive hyperglycemia, further contributing to insulin resistance
