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Compared with the NCD group, HFD-fed mice displayed significant enrichment of pro-inflammatory pathways, including cytokinecytokine receptor interaction (NES = 1.712, P = 0.0015), chemokine signaling (NES = 1.895, P = 0.0016), NF-B signaling (NES = 1.609, P = 0.0049), and Toll-like receptor signaling (NES = 1.806, P = 0.0016), confirming a strong activation of innate and adaptive immune processes in adipose tissue (Fig
22,24,29 We assessed the two nonrandomized studies with the ROBINS-I tool (ESM eFigs
It inhibits the production of pro-inflammatory cytokines (TNF-alpha, IL-6, IL-12) and modulates macrophage polarization towards the M2 phenotype (reparative/anti-inflammatory)
