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Increasing the expression of HO-1 or improving the function of nitrergic mechanisms through experimental approaches protects against the development of gastroparesis or restores gastric emptying in diabetic mice and rats, respectively [58]

1 after PCR-amplification

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Treg cell-specific mitochondria Opa1 deletion or lysosome Flcn deletion phenocopies and this interplay reprograms Treg cell differentiation and functions with direct consequences for immune tolerance (66)