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Molecular docking analyses did not involve biological replicates but were performed as computational technical replicates to validate binding interactions
The net result: retatrutide's triple mechanism produces more GI disruption than either GLP-1 alone (semaglutide) or GLP-1 plus GIP (tirzepatide)
Insulin secretion from oral glucose intake is greater than insulin secretion from IV glucose, termed the incretin effect [13]
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