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every current supply is compounded or research-grade, and its human evidence base is older and mostly about pediatric growth-hormone deficiency
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Alteration of several metabolic enzymes and pathways that affect KG abundance and KGDD activity have been reported in cancer, such as mutations of the genes encoding isocitrate dehydrogenase 1 and 2 (IDH1 and IDH2), changes in glutaminolysis that deplete KG, and alterations in fumarate hydratase and succinate dehydrogenase that inhibit KGDD activity 11
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doi:10.1186/s12974-020-01752-1 126
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