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Oxidative stresscharacterized by excessive production of reactive oxygen species (ROS)is both a cause and consequence of chronic inflammation, creating a self-perpetuating cycle of tissue damage
MIC19 is not only a key member of mitochondrial cristae junctions, responsible for the organisation of mitochondrial inner and outer membrane contacts, as well as for the architecture of respiratory complexes and the mitochondrial proton pump, but also plays a prominent role in ERMCSs, where it enables the transfer of lipid precursors for the biosynthesis of the key mitochondrial lipid cardiolipin 68 , therefore connecting mitochondrial cristae organisation with the generation of membrane curvature and control of mitochondrial function afforded by cardiolipin 11
It supports the stability of the liposomes and allows for an extended shelf life
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