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Acute kidney injury in diabetes In T1DM mice with ischemiareperfusion-induced acute kidney injury (AKI), ESC (50, 100 mg/kg), ESC enhanced mitophagy in renal tubular epithelial cells (NRK-52E) subjected to hypoxia-reoxygenation injury, characterized by increased levels of PINK1 and Parkin (mitophagy markers), reduced oxidative stress via the Nrf2/Keap1 pathway, cell apoptosis, and maintained mitochondrial integrity (Dagar et al
Ch.2.2.2.1 (eds Picardo, M
CHR-6494 (500 nM) produces a mitotic catastrophe with abnormal morphology of the mitotic spindle and centrosome amplification, and upregulates the spindle assembly checkpoint protein BUB1 and the marker of mitotic arrest cyclin B1 [1]
Singh, M.D., a family and obesity medicine physician at Henry Ford Health
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