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Consequently, it seems there are important combinations of distinct signaling properties to the biased agonism of TZPD, which further may account for the promising efficacy of TZPD in damaged cells including cardiomyocytes
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Beyond defining a mitochondrial mechanism for fuel switching, this work provides a conceptual landscape for future investigation of metabolic adaptation across diverse physiological contexts, including exercise, distinct fasting paradigms, and hormone-driven metabolic regulation, and offers new insight into the determinants of responsiveness to GLP1R agonist-based anti-obesity therapies
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