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This pattern reflects the transition from acute anti-inflammatory effects to slower structural regeneration

Furthermore, RA is also thought to be driven by a generation of neoantigens through post-translational modifications of proteins, most commonly by citrullination (also known as deimination)the replacement of the ketamine group in arginine with a ketone group, which effectively removes the positive charge of arginine at neutral pH and converts arginine to the non-coded amino acid citrulline [208, 209]

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This mechanism of action probably plays a major role, as inflammations are causally involved in the development of nerve damage in most neurological diseases