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GLP-1, GIP, exendin-4 and liraglutide had suppressive effects on oxLDL-induced foam cell formation, which is linked with the suppression of infiltrated macrophage foam cells in atherosclerotic plaques, resulting in repressed atherosclerotic plaque formation
Regular follow-up appointments allow for progress monitoring and adjustments as necessary
Author Affiliations | Correspondence | Abstract | Citation | Keywords | PDF Author Affiliations Departments of Anesthesiology and Medicine, Columbia University Medical Center, New York, New York Department of Medicine, Queen Elizabeth Hospital, Birmingham, United Kingdom Address correspondence to: Monica Goldklang, MD Columbia University Medical Center 630 West 168th Street, P&S 12-402 New York, NY 10032 Email: [email protected] Abstract This article serves as a CME-available, enduring material summary of the following COPD9 USA presentations: Overview of Lung Injury in COPD: Types and Key Questions Presenter: Caroline Owen, MD, PhD Emphysema as a Disease of Deficient Tissue Repair/Maintenance Presenter : Rubin Tuder, MD Article References Running Head: Pathophysiology of Emphysema Abbreviations : chronic obstructive pulmonary disease
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