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Costantini, D
A causal link between the higher anorexigenic hormone concentrations and post-RYGB appetite control has been supported by studies, where ad libitum food intake is increased after inhibition of hormone secretions by the somatostatin analogue octreotide in humans [10, 14] and rats [15] or after combined inhibition of GLP-1 and PYY actions in humans [16]
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Instead, this drug interrupts signalling that could affect appetite in parts of the central nervous system that are not behind the blood-brain barrier
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