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Myosin light chain kinase is central to smooth muscle contraction and required for gastrointestinal motility in mice
The lack of statistically significant reduction in infarct volume, despite a numerical decrease, suggests that DSIPs primary benefit in this model might be more related to functional neurorecovery and neural network plasticity rather than a direct, massive reduction of necrotic tissue [1]
This is why improving bile flow is a direct methylation intervention, not just a digestive one
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