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In the face of controversies and challenges, it is essential to establish a more efficient translational interface between science, technology, and clinical practice, driving mitochondrial metabolic reprogramming therapy from a potential pathway to clinical reality. Conclusion Mitochondrial metabolic reprogramming plays a central role in the therapeutic failure of CRC
Modulation of cellular disulfide-bond formation and the ER redox environment by feedback regulation of Ero1
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Participates in metabolic processes important for normal brain and nervous system function.*
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